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To date, no symptomatic treatment is available for patients with vascular cognitive impairment VCI. In the proof-of-principle study Symptomatic Treatment of Vascular Cognitive Impairment STREAM-VCI, we investigated whether a single dose of a monoaminergic drug methylphenidate improves executive functioning and whether a single dose of a cholinergic drug galantamine improves memory in VCI patients. Methylphenidate was well tolerated while galantamine produced gastrointestinal side effects in a considerable number of patients. Galantamine did not improve memory or executive dysfunction. Results might be influenced by the considerable amount of side effects seen. Vascular cognitive impairment VCI is an important cause of cognitive decline and dementia 1.

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Treating the symptoms of AD can provide patients with comfort, dignity and independence for a longer period of time and can encourage and assist their caregivers as well. These drugs are prescribed for the treatment of mild to moderate AD. They may help delay or prevent symptoms from becoming worse for a limited time and may help control some behavioral symptoms. Scientists do not yet fully understand how cholinesterase inhibitors work to treat AD, but current research indicates that they prevent the breakdown of acetylcholine, a brain chemical believed to be important for memory and thinking. As AD progresses, the brain produces less and less acetylcholine; therefore, cholinesterase inhibitors may eventually lose their effect. Because all four work in a similar way, it is not expected that switching from one of these drugs to another will produce significantly different results. However, an AD patient may respond better to one drug than another. It is prescribed for the treatment of moderate to severe AD.

Background: The metabolic syndrome MetS is an obesity-associated disorder of pandemic proportions and limited treatment options. Oxidative stress, low-grade inflammation and altered neural autonomic regulation, are important components and drivers of pathogenesis. Here we examined the effects of galantamine on oxidative stress in parallel with inflammatory and cardio-metabolic parameters in subjects with MetS. Oxidative stress, including superoxide dismutase SOD, catalase CAT, and glutathione peroxidase activities, lipid and protein peroxidation, and nitrite levels were analyzed before and at the end of the treatment. In addition, plasma cytokine and adipokine levels, insulin resistance HOMA-IR and other relevant cardio-metabolic indices were analyzed.

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Reminyl is an Alzheimer's treatment derived from the bulbs of the daffodil, Narcissus pseudonarcissus. It is believed that neurons producing the neurotransmitter acetylcholine degenerate in the brains of patients with Alzheimer's disease. Reminyl works to increase the concentration of acetylcholine by blocking the action of acetylcholinesterase, an enzyme that catalyzes the hydrolysis break down of acetylcholine. An estimated four million Americans have Alzheimer's disease -- a progressive loss of cognitive function so severe that it interferes with an individual's ability to function. The number is expected to grow to 14 million by the middle of the next century.

Slow heart rate This drug may cause a slow heart rate and fainting. This happens more often in people with heart problems. Risk of worse lung disease Tell your doctor if you have a history of asthma or other lung diseases. Galantamine may make these conditions worse, so it needs to be used with caution. Generic drugs usually cost less.

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Alzheimer disease, mild to moderate Range: 16 to 24 mg daily in 2 divided doses. Range: 16 to 24 mg once daily. May decrease to 8 mg twice daily if the target dose is not tolerated. Range: 16 to 24 mg daily in 2 divided reminyl 8 mg tablets Ref. Dementia with Lewy bodies and Parkinson disease dementia off-label use: Note: In patients with mild or moderate dementia with Lewy bodies, consider galantamine therapy only in patients who do not tolerate donepezil and rivastigmine Ref.

Takeda Pharmaceutical Company Limited. Approval details may vary by country. Medicines have adverse reactions risks as well as efficacies benefits. It is important to minimize adverse reactions and maximize efficacy. To obtain a better therapeutic response, patients should understand their medication and cooperate with the treatment. This medicine delays the progression of symptoms of dementia such as memory impairment forgetfulness, disorientation problems in cognition of time and place and difficulty in making judgments, by preventing decrease of acetylcholine, a neurotransmitter in brain, via inhibition of the action of an enzyme catalyzing acetylcholine degradation and by facilitating nerve signaling.

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